I don't have time for this. One of the fundamental disfunctions of T1D is that alpha cells release excessive glucagon in response to eating, largely due to reflexes in intestinal cells that usually stimulate insulin and glucagon in balance in non T1Ds, but it T1Ds there is no insulin to match. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2453684/ A major reason people take synthetic amylin is that it blocks the disfunctional glucagon release that develops in T1Ds over the first 4-5 years. Also T1Ds alpha cells start to lose their ability to release glucagon to alleviate hypoglycemia. There have been studies showing that eating large volumes of lettuce can trigger a reflexive release of glucagon that raises blood sugar significantly (up to 100 points) despite fewer than 10 grams of carbs.